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radon - documentation of exposure histories for Iowa study



Jim Muckerheide,



Ray Johnson may not be an epidemiologist, but the point was that Ray knows a 

few things about radon exposure assessment.  As far as Phillipe Dupont, he 

is a frequent poster of emails on your Radiation and Science listserv and an 

known anti LNT proponent.



If the Iowa study did not find an association, then they may have said the 

power was insufficient to detect an association as is likely the case in 

many studies with poor exposure ascertainment.  But, the Iowa study did find 

an association so therefore the sample size was of sufficient power to 

detect an association.



I saw your "explanation" given to Les.  It was nonsensical to me as it 

apparently was to Les.  As far as your misrepresentations, I am not sure 

where to start.  The latest item you have tried to mislead people with is 

that the Iowa Study did not obtain individual exposure histories for each 

subject. Your insistence is that because someone lives in Iowa, that have to 

be exposed to high radon levels.  As Les, pointed out.  In the Iowa study 

half, the people in the study were exposed to less than 4 pCi/L (average 

15--year exposure).  Their exposure data was log normally distributed (that 

means they had a lot of data for lower exposed individuals).



The Iowa study did an incredible job documenting individual exposure 

including many supporting papers as Les has pointed out to me.  On one hand 

we have your misrepresentation and on the other hand we have these detailed 

papers below laying out how they accounted for radon exposure.



------------------------------------------

Citation: Field RW, Smith BJ, Brus CP, Lynch CF, Neuberger JS, Steck DJ, 

Retrospective temporal and spatial mobility of adult Iowa women., Risk Anal 

18: 5, 575-84, Oct, 1998.





Abstract

Human exposure assessments require a linkage between toxicant concentrations 

in occupied spaces and the receptor's mobility pattern. Databases reporting 

distinct populations' mobility in various parts of the home, time outside 

the home, and time in another building are scarce. Temporal longitudinal 

trends in these mobility patterns for specific age and gender groups are 

nonexistent. This paper describes subgroup trends in the spatial and 

temporal mobility patterns within the home, outside the home, and in another 

building for 619 Iowa females that occupied the same home for at least 20 

years. The study found that the mean time spent at home for the participants 

ranged from a low of 69.4% for the 50-59 year age group to a high of 81.6% 

for the over 80-year-old age group. Participants who lived in either one- or 

two- story homes with basements spent the majority of their residential 

occupancy on the first story. Trends across age varied for other subgroups 

by number of children, education, and urban/rural status. Since all of these 

trends were nonlinear, they indicate that error exists when assuming a 

constant, such as a 75% home occupancy factor, which has been advocated by 

some researchers and agencies. In addition, while aggregate data, such as 

presented in this report, are more helpful in deriving risk estimates for 

population subgroups, they cannot supplant good individual-level data for 

determining risks.



------------------------------------------------------------



Medline ID: 21270976



Citation: Field RW, Steck DJ, Smith BJ, Brus CP, Fisher EF, Neuberger JS, 

Lynch CF, The Iowa radon lung cancer study--phase I: Residential radon gas 

exposure and lung cancer., Sci Total Environ 272: 1-3, 67-72, May14, 2001.



Abstract

Exposure to high concentrations of radon (222Rn) progeny produces lung 

cancer in both underground miners and experimentally-exposed laboratory 

animals. The goal of the study was to determine whether or not residential 

radon exposure exhibits a statistically significant association with lung 

cancer in a state with high residential radon concentrations. A 

population-based, case-control epidemiologic study was conducted examining 

the relationship between residential radon gas exposure and lung cancer in 

Iowa females who occupied their current home for at least 20 years. The 

study included 413 incident lung cancer cases and 614 age-frequency-matched 

controls. Participant information was obtained by a mailed-out questionnaire 

with face-to-face follow-up. Radon dosimetry assessment consisted of five 

components: (1) on-site residential assessment survey; (2) on-site radon 

measurements; (3) regional outdoor radon measurements; (4) assessment of 

subjects' exposure when in another building; and (5) linkage of historic 

subject mobility with residential, outdoor, and other building radon 

concentrations. Histologic review was performed for 96% of the cases. 

Approximately 60% of the basement radon concentrations and 30% of the first 

floor radon concentrations of study participants' homes exceeded the US 

Environmental Protection Agency action level of 150 Bq m(-3) (4 pCi l(-1)). 

Large areas of western Iowa had outdoor radon concentrations comparable to 

the national average indoor value of 55 Bq m(-3) (1.5 pCi l(-1)). Excess 

odds of 0.24 (95% CI = -0.05-0.92) and 0.49 (95% CI = 0.03-1.84) per 11 

WLM(5-19) were calculated using the continuous radon exposure estimates for 

all cases and live cases, respectively. Slightly higher excess odds of 0.50 

(95% CI = 0.004-1.80) and 0.83 (CI = 0.11-3.34) per 11 WLM(5-19) were noted 

for the categorical radon exposure estimates for all cases and the live 

cases. A positive association between cumulative radon gas exposure and lung 

cancer was demonstrated using both categorical and continuous analyses. The 

risk estimates obtained in this study indicate that cumulative radon 

exposure presents an important environmental health hazard.



----------------------------------------------------------------

  Environmental Health Perspectives Volume 107, Number 11, November 1999





Intercomparison of Retrospective Radon Detectors

R. William Field,1 Daniel J. Steck,2 Mary Ann Parkhurst,3 Judy A. Mahaffey,3 

and Michael C.R. Alavanja4



1Department of Epidemiology, College of Public Health, University of Iowa, 

Iowa City, Iowa, USA

2Physics Department, St. John's University, Collegeville, Minnesota, USA

3Pacific Northwest National Laboratory, Richland, Washington, USA

4National Cancer Institute, Bethesda, Maryland, USA





Abstract

We performed both a laboratory and a field intercomparison of two novel 

glass-based retrospective radon detectors previously used in major radon 

case-control studies performed in Missouri and Iowa. The new detectors 

estimate retrospective residential radon exposure from the accumulation of a 

long-lived radon decay product, 210Pb, in glass. The detectors use track 

registration material in direct contact with glass surfaces to measure the 

-emission of a 210Pb-decay product, 210Po. The detector's track density 

generation rate (tracks per square centimeter per hour) is proportional to 

the surface -activity. In the absence of other strong sources of -emission 

in the glass, the implanted surface -activity should be proportional to the 

accumulated 210Po, and hence to the cumulative radon gas exposure. The goals 

of the intercomparison were to a) perform collocated measurements using two 

different glass-based retrospective radon detectors in a controlled 

laboratory environment to compare their relative response to implanted 

polonium in the absence of environmental variation, b) perform collocated 

measurements using two different retrospective radon progeny detectors in a 

variety of residential settings to compare their detection of 

glass-implanted polonium activities, and c) examine the correlation between 

track density rates and contemporary radon gas concentrations. The 

laboratory results suggested that the materials and methods used by the 

studies produced similar track densities in detectors exposed to the same 

implanted 210Po activity. The field phase of the intercomparison found 

excellent agreement between the track density rates for the two types of 

retrospective detectors. The correlation between the track density rates and 

direct contemporary radon concentration measurements was relatively high, 

considering that no adjustments were performed to account for either the 

residential depositional environment or glass surface type. Preliminary 

comparisons of the models used to translate track rate densities to average 

long-term radon concentrations differ between the two studies. Further 

calibration of the retrospective detectors' models for interpretation of 

track rate density may allow the pooling of studies that use glass-based 

retrospective radon detectors to determine historic residential radon 

exposures. Key words: case-control studies, dose-response relationship 

(radiation), epidemiologic methods, epidemiologic studies, lung neoplasms, 

radon, radon progeny, smoking. Environ Health Perspect 107:905-910 (1999). 

[Online 15 October 1999]

http://ehpnet1.niehs.nih.gov/docs/1999/107p905-910field/abstract.html

Address correspondence to R.W. Field, College of Public Health, Department 

of Epidemiology, N222 Oakdale Hall, University of Iowa, Iowa City, IA 52242 

USA. Telephone: (319) 335-4413. Fax: (319) 335-4747. E-mail: 

bill-field@uiowa.edu

We thank J. Huber, C. Greaves, G. Buckner, J. Jesse, and E. Berger for their 

assistance with data collection. We also thank C. Lynch and J. Lubin for 

their reviews of previous versions of this manuscript.



NCI contract 263-MQ-820009 and NIEHS grant P30 ESO5605 supported this 

research. This report is solely the responsibility of the authors and does 

not necessarily reflect the official views of the NCI, NIEHS, or NIH.



----------------------------------------------------------------

Medline ID: 99122934



Citation: Steck DJ, Field RW, Lynch CF, Exposure to atmospheric radon., 

Environ Health Perspect 107: 2, 123-7, Feb, 1999.

Address: Department of Physics



Abstract

We measured radon (222Rn) concentrations in Iowa and Minnesota and found 

that unusually high annual average radon concentrations occur outdoors in 

portions of central North America. In some areas, outdoor concentrations 

exceed the national average indoor radon concentration. The general spatial 

patterns of outdoor radon and indoor radon are similar to the spatial 

distribution of radon progeny in the soil. Outdoor radon exposure in this 

region can be a substantial fraction of an individual's total radon exposure 

and is highly variable across the population. Estimated lifetime effective 

dose equivalents for the women participants in a radon-related lung cancer 

study varied by a factor of two at the median dose, 8 mSv, and ranged up to 

60 mSv (6 rem). Failure to include these doses can reduce the statistical 

power of epidemiologic studies that examine the lung cancer risk associated 

with residential radon exposure.

-----------------------------------



Medline ID: 99005221

Citation: Fisher EL, Field RW, Smith BJ, Lynch CF, Steck DJ, Neuberger JS, 

Spatial variation of residential radon concentrations: the Iowa Radon Lung 

Cancer Study., Health Phys 75: 5, 506-13, Nov, 1998.



Abstract

Homeowners and researchers frequently estimate the radon concentrations in 

various areas of the home from a single radon measurement often performed in 

the home's basement. This study describes the spatial variation of radon 

concentrations both between floors and between rooms on the same floor. The 

geometric mean basement and first floor radon concentrations for one-story 

homes were 13.8% and 9.0% higher, respectively, as compared to their 

counterparts in two-story homes. The median first floor/basement ratio of 

radon concentrations for one-story homes was 0.60. The median ratios between 

first floor/basement and second floor/basement for two-story homes were 0.51 

and 0.62, respectively. The mean coefficient of variation for detectors 

placed on the same floor was 9.5%, which was only 2.6% higher than the mean 

coefficient of variation found for collocated (duplicate) quality control 

detectors. The wide individual variations noted in radon concentrations 

serve as a reminder of the importance of performing multiple radon 

measurements in various parts of the home when estimating home radon 

concentrations.



-------------------------------------------------------

Field, R.W., Lynch, C.F., Steck, D.J. and Fisher, E.F.. Dosimetry Quality 

assurance: the Iowa residential radon lung cancer study. Radiation 

Protection Dosimetry. 78(4): 295-303, 1998



-----------------------------------------------

Medline ID: 96384408

Citation: Field RW, Steck DJ, Lynch CF, Brus CP, Neuberger JS, Kross BC, 

Residential radon-222 exposure and lung cancer: exposure assessment 

methodology., J Expo Anal Environ Epidemiol 6: 2, 181-95, Apr-Jun, 1996.



Abstract

Although occupational epidemiological studies and animal experimentation 

provide strong evidence that radon-222 (222Rn) progeny exposure causes lung 

cancer, residential epidemiological studies have not confirmed this 

association. Past residential epidemiological studies have yielded 

contradictory findings. Exposure misclassification has seriously compromised 

the ability of these studies to detect whether an association exists between 

222Rn exposure and lung cancer. Misclassification of 222Rn exposure has 

arisen primarily from: 1) detector measurement error; 2) failure to consider 

temporal and spatial 222Rn variations within a home; 3) missing data from 

previously occupied homes that currently are inaccessible; 4) failure to 

link 222Rn concentrations with subject mobility; and 5) measuring 222Rn gas 

concentration as a surrogate for 222Rn progeny exposure. This paper examines 

these methodological dosimetry problems and addresses how we are accounting 

for them in an ongoing, population-based, case-control study of 222Rn and 

lung cancer in Iowa.



--------------------------------------------



Medline ID: 20329535

Citation: Field RW, Steck DJ, Smith BJ, Brus CP, Fisher EL, Neuberger JS, 

Platz CE, Robinson RA, Woolson RF, Lynch CF, Residential radon gas exposure 

and lung cancer: the Iowa Radon Lung Cancer Study., Am J Epidemiol 151: 11, 

1091-102, Jun1, 2000.



Abstract

Exposure to high concentrations of radon progeny (radon) produces lung 

cancer in both underground miners and experimentally exposed laboratory 

animals. To determine the risk posed by residential radon exposure, the 

authors performed a population-based, case-control epidemiologic study in 

Iowa from 1993 to 1997. Subjects were female Iowa residents who had occupied 

their current home for at least 20 years. A total of 413 lung cancer cases 

and 614 age-frequency-matched controls were included in the final analysis. 

Excess odds were calculated per 11 working-level months for exposures that 

occurred 5-19 years (WLM(5-19)) prior to diagnosis for cases or prior to 

time of interview for controls. Eleven WLM(5-19) is approximately equal to 

an average residential radon exposure of 4 pCl/liter (148 Bq/m3) during this 

period. After adjustment for age, smoking, and education, the authors found 

excess odds of 0.50 (95% confidence interval: 0.004, 1.81) and 0.83 (95% 

percent confidence interval: 0.11, 3.34) using categorical radon exposure 

estimates for all cases and for live cases, respectively. Slightly lower 

excess odds of 0.24 (95 percent confidence interval: -0.05, 0.92) and 0.49 

(95 percent confidence interval: 0.03, 1.84) per 11 WLM(5-19) were noted for 

continuous radon exposure estimates for all subjects and live subjects only. 

The observed risk estimates suggest that cumulative ambient radon exposure 

presents an important environmental health hazard.



Don Smith







----------------------------------------------

>From: "Jim Muckerheide" <jmuckerheide@cnts.wpi.edu>

>To: "Rad health" <healthrad@hotmail.com>, <lescrable@hotmail.com>, 

><jkotton@usgs.gov>

>CC: <radsafe@list.vanderbilt.edu>

>Subject: RE: radon - and tackling the issues

>Date: Tue, 15 Jan 2002 04:11:47 -0500

>

You haven't read, or perhaps understood, the problem. See the

explanation provided to Les. If you think something is "misleading," let me 

know. It's just my take on the failure of radon case-control studies.



And why Iowa is the worst place, despite Field's efforts to relate a

lifetime of radon exposure to house measurements. It just can't work,

and the study is too small to produce a credible, replicable result.



Ray's a great guy, but his forte is HP training, and communication. If

you want an expert on radon health effects try Philippe Duport at U

Ottawa, with 40 years originally focused on the French miners and the

industry, then to Canada, as a regulator, and nor at the university. No 

adverse radon effects at low-dose, low dose-rate, at much higher than





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