Thank you, what you presented is a good
start. I was wondering, if the years of exposure had something to do with
it.
H. Dean Chaney, CHP URS Corp. Sacramento, CA (916) 679-2086
"In science there is only physics; everything else is stamp
collecting."
--Ernest Rutherford
----- Original Message -----
Sent: Saturday, July 27, 2002 7:09
AM
Subject: RE: RESRAD Information
Mr.
Chaney,
I
am a former workshop instructor for the RESRAD Family of Codes, and
while I no longer work for the code developers at ANL, I have followed
the code's recent developments very closely due to my current work involving
uses of the RESRAD code.
I
assume that when you mean dose-based risk values, you are converting the doses
calculated by RESRAD to a risk by using a dose-to-risk conversion factor
(e.g., dose-to-risk conversion factor of
4E-7 latent cancer fatalities per mrem). If that is the case, then
the first reason that the risk values calculated by RESRAD
would be different than the risk-converted dose values is because RESRAD
uses FGR 13 numbers to calculate risk directly from external exposure or
intakes, rather than applying a single
dose-to-risk conversion factor. Thus, there will be a significant
variation in the dose/risk ratios for different radionuclides depending on the
dominant pathway and/or affected organ (for intake-dominated
exposures).
Also, and perhaps more importantly, the default exposure duration for
dose-based results is one year (results are presented as mrem/year).
However, the default exposure duration for risk-based calculations is 30 years
AFTER the user-specified time at which the exposure is assumed to start (but
the user can change this default to one year or any other duration of
interest). The reason to use a multi-year exposure duration is to bring
the calculations into closer agreement with the EPA methodology for
calculating risks from exposure to chemical carcinogens. This usually
has the apparent effect of increasing the calculated risk numbers
significantly above the values estimated based on a direct conversion from
annual dose to risk using a dose/risk conversion factor. Note that the
increase is not always around 30 times higher, since RESRAD takes into account
decay, ingrowth and transport of radionuclides to integrate the concentrations
in the various media over the exposure duration period. For long-lived,
immobile nuclides with no significant ingrowth relative to the exposure
duration period, the values will be very close to 30 times higher.
However, for shorter-lived or very mobile nuclides, the discrepancy will
likely be less than a factor of 30, unless the concentrations are increasing
over the 30-year period, as might be the case for the groundwater pathway
doses immediately following breakthrough to the saturated
zone.
Hope
this helps. Please let me know if you have need me to clarify or expand
on my response or assist you with any other RESRAD-related
issues.
Ernesto Faillace, Eng.D,
CHP Nuclear Engineer/Health Physicist Tetra Tech NUS 900 Trail
Ridge Rd Aiken, SC 29803 (803) 649-7963 x303 (803) 642-8454 (fax)
faillacee@ttnus.com
Not being a regular user of RESRAD the
following question has come up and I could not find a suitable answer in the
RESRAD manuals:
When RESRAD (latest version) provides
a dose based risk value for several isotopes it comes up with different
risk values. Does anyone know why dose based risk values, assuming the
doses are TEDE, would be different?
H. Dean Chaney, CHP URS Corp. Sacramento,
CA (916) 679-2086
"In science there is only physics; everything
else is stamp
collecting."
--Ernest Rutherford
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