[ RadSafe ] Is this the beginning of the end?
JPreisig at aol.com
JPreisig at aol.com
Tue Jul 8 13:46:59 CDT 2014
Nice comments. Many different Cancers, many different production
mechanisms. Unmapped parts of Human DNA may have information. Junk DNA may
be where you install the telepathic ability option, other things etc.
Cancer is a response to a carcinogen, irritant, insult etc. Is the body's
response to cancer programmed in our DNA??? Find out.
We treat Cancer many ways: radiation, chemotherapy, surgery,
The cancer induction problem sound like the monkeys producing great
novels using typewriters. See Kittel's Thermal Physics for this great novel
Another way to cure the cancer is to repair structural damage and
sucking out there carcinogen etc.
Clayton, solve the questions you have posed and you probably have
cured cancer. See you on the Nobel prize podium. Keep cranking on the problem.
In a message dated 7/7/2014 5:22:28 P.M. Eastern Daylight Time,
clayton.bradt at health.ny.gov writes:
This is not exactly on topic but ...
I am not a biologist, nor do I play one on TV, but I would appreciate
anyone's thoughts on this problem I've been grappling with on the subject of
radiation induced cancer, and indeed all cancer induction:
Are cancer cells just simpler, stripped down versions of the somatic cell
from which they are derived? If not, it is difficult to understand how
random damage to a somatic cell could result in a cancer cell, as opposed to
say another kind of somatic cell. No matter how long and involved the actual
transformation process is, the fact is that we start with random DNA
damage and end up with a new kind of cell. Now, random damage to a Ford doesn't
turn it into a Ferrari (or even a Chevy). Nor will random damage to a liver
cell turn it into a cardiac cell --- will it? Whence, then cancer cells?
Certain cancers have been traced to viral agents: cervical cancer from
HPV, and liver cancer from hepatitis C virus. In such cases viral gene
sequences are spliced into the somatic cells' DNA resulting them transforming into
tumor cells. In other words the damage is not random, but rather specific
to producing a viable cancer cell. Presumably the specific genes that the
virus splices into the host cell have evolved over evolutionary time scales
far exceeding the lifetimes of the host organism or the virus.
It is reasonable to apply a similar paradigm to other cancers. Somehow,
the specific gene sequences enabling an ordinary somatic cell to "go wild",
as it were, must become spliced into the cell's DNA. Any old random
rearrangement isn't enough.
If radiation damaged somatic cells must somehow perform this sort of
genetic rearrangement, it is difficult to see how there could be enough time to
do so within the short span of the organism's life.
Is it possible that as yet unidentified viral agents are in fact
responsible for all cancers?
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