[ RadSafe ] Is this the beginning of the end?

[JPreisig at aol.com]

Clayton/Radsafe:
 
     Nice comments.  Many different Cancers, many  different production 
mechanisms.   Unmapped parts of Human DNA may  have information.  Junk DNA may 
be where you install the telepathic ability  option, other things etc.  
Cancer is a response to a carcinogen, irritant,  insult etc.  Is the body's 
response to cancer programmed in our  DNA???  Find out.
 
    We treat Cancer many ways: radiation, chemotherapy,  surgery, 
immunotherapy etc.
 
    The cancer induction problem sound like the monkeys  producing great 
novels using typewriters.  See Kittel's Thermal Physics for  this great novel 
scenario.
 
    Another way to cure the cancer is to repair structural  damage and 
sucking out there carcinogen etc.
 
    Clayton, solve the questions you have posed and you  probably have 
cured cancer.  See you on the Nobel prize podium.  Keep  cranking on the problem.
 
 
     Joe Preisig
 
 
 
 
In a message dated 7/7/2014 5:22:28 P.M. Eastern Daylight Time,  
clayton.bradt at health.ny.gov writes:


This  is not exactly on topic but ...

I am not a biologist, nor do I play one  on TV, but I would appreciate 
anyone's thoughts on this problem I've been  grappling with on the subject of 
radiation induced cancer, and indeed all  cancer induction:

Are cancer cells just simpler, stripped down versions  of the somatic cell 
from which they are derived?  If not, it is difficult  to understand how 
random damage to a somatic cell could result in a cancer  cell, as opposed to 
say another kind of somatic cell. No matter how long and  involved the actual 
transformation process is, the fact is that we start with  random DNA 
damage and end up with a new kind of cell. Now, random damage to a  Ford doesn't 
turn it into a Ferrari (or even a Chevy). Nor will random damage  to a liver 
cell turn it into a cardiac cell --- will it?  Whence, then  cancer cells?

Certain cancers have been traced to viral agents:  cervical cancer from 
HPV, and liver cancer from hepatitis C virus. In such  cases viral gene 
sequences are spliced into the somatic cells' DNA resulting  them transforming into 
tumor cells. In other words the damage is not random,  but rather specific 
to producing a viable cancer cell.  Presumably the  specific genes that the 
virus splices into the host cell have evolved over  evolutionary time scales 
far exceeding the lifetimes of the host organism or  the virus.

It is reasonable to apply a similar paradigm to other  cancers. Somehow, 
the specific gene sequences enabling an ordinary somatic  cell to "go wild", 
as it were, must become spliced into the cell's DNA. Any  old random 
rearrangement isn't enough.

If radiation damaged somatic  cells must somehow perform this sort of 
genetic rearrangement, it is difficult  to see how there could be enough time to 
do so within the short span of the  organism's life.

Is it possible that as yet unidentified viral agents  are in fact 
responsible for all  cancers?


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